Re: No danger at all

You're in over your head. Here is a paper describing how SSRI's improve NK cell function in HIV. Most psychiatric drugs work by modulating the immune system in one way or another. The mechanism of action of psychiatric drugs is purposefully misleading by the drug companies. The whole psychiatric disease theory is a joke, most brain and behavioral problems are most likely the result of persistent ongoing infections.

Saying that meditation can cure every single variety of these infections that manifest as "psychiatric" diseases is nonsense.

I do think that meditation may cure mild to moderate depression, as well as other illnesses. It will also help a lot in times that it does not cure. But that doesn't mean it is good for every single thing out there. Geez, be open minded. You are the close minded one, I don't see how you don't understand that.


Evans DL, Lynch KG, Benton T, Dubé B, Gettes DR, Tustin NB, Lai JP, Metzger D, Douglas SD.

Department of Psychiatry, Joseph J. Stokes Research Institute of The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA. psych@mail.med.upenn.edu
Abstract

BACKGROUND: Natural killer (NK) cells play an important role in innate immunity and are involved in the host defense against human immunodeficiency virus (HIV) infection. This study examines the potential role of three underlying regulatory systems that have been under investigation in central nervous system research as well as immune and viral research: serotonin, neurokinin, and glucocorticoid systems. METHODS: Fifty-one HIV-seropositive subjects were recruited to achieve a representative sample of depressed and nondepressed women. The effects of a selective serotonin reuptake inhibitor (SSRI), a substance P (SP) antagonist, and a glucocorticoid antagonist on NK cell function were assessed in a series of ex vivo experiments of peripheral blood mononuclear cells from each HIV-seropositive subject. RESULTS: Natural killer cell cytolytic activity was significantly increased by the SSRI citalopram and by the substance P antagonist CP-96345 relative to control conditions; the glucocorticoid antagonist, RU486, showed no effect on NK cytotoxicity. Our results suggest that the effects of the three agents did not differ as a function of depression. CONCLUSIONS: Our findings provide evidence that NK cell function in HIV infection may be enhanced by serotonin reuptake inhibition and by substance P antagonism. It remains to be determined if HIV-related impairment in not only NK cytolytic activity but also NK noncytolytic activity can be improved by an SSRI or an SP antagonist. Clinical studies are warranted to address these questions and the potential roles of serotonergic agents and SP antagonists in improving NK cell immunity, delaying HIV disease progression, and extending survival with HIV infection.

PMID: 17945197

Based on my research and opinion, IL-4 is more directly implicated as problematic in human Lyme infection than IL-10. Thus, even if it stimulates IL-10 production, suppression of IL-4 by SSRI's may have a net positive impact:

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