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Heart Attacks, Strokes, Cancers and Tumors Explained!
Elijah Views: 2,423
Published: 19 years ago
Status:       RN [Message recommended for CureZone Newsletter!]

Heart Attacks, Strokes, Cancers and Tumors Explained!

Heart attacks, strokes, cancers, tumors and other gene related diseases are all EXPLAINED in this brief essay or report. (Ed. Note: Perhaps someone out there will read this and send it to someone like Björn Nordenström, M.D., Ph.D., former chairman of the Department of Radiology at Karolinska Hospital and chairman of the selection committee that picks the Nobel Laureate for Physiology of Medicine!)

This truly explains how drugs and various DNA transferring bacteria are the major cause of heart attacks, strokes, and many cancers, gene related diseases. Primarily – any of those diseases emerge following exposure to a chemical, carcinogen, drug, or even a food which induces an upset of the normal ecological balance of the intestinal flora. In reality, smoking, or any of the above can commonly results in a general suppression of the host’s immune system. I will explain what causes all the heart attacks and strokes, first. Exposure to various environmental toxins often causes people to become susceptible to Candida; chemicals, food additives , and poor nutrition also enable Candida to thrive. This type of bacteria is also able to transform itself, growing root-like, rhizoid like legs and producing toxins that further lower a person’s immunity. In other words there are many things that can (and usually do) result in Candida overgrowth. More specifically, cortisone, prednisone, and other steroid-type drugs are known to cause the opportunistic Candida to thrive. These drugs, and other drugs and chemicals, paralyze the body’s defense systems, just as birth control pills and various estrogens are known to upset hormonal balances within the body, and thereby these also contribute to the abnormal growth of excessive Candida. (Note: some bacteria in the clostridium family also break down fats into the minor estrogens, otherwise known as “impeded estrogens.”) There are also various contributing factors such as the western diet being typically high in Sugar and other refined carbohydrates, which readily promote the growth of drug-resistant Candida and other high-energy, drug-resistant bacterium. Candida also “impair immune functioning by directly and negatively impacting the helper-suppresser ratio of T lymphocytes,” (according to Charles R. Caulfield in his article "Natural Therapies For Candidiasis Infections” in the San Francisco Sentinel, August 27, 1992.) When it emerges, the organism progressively spreads out from the lower bowel to colonize the entire digestive tract, successively including the stomach, (especially in cases of low or no stomach acid) and from there into the throat, mouth, and even the nasal passages. From there, these microorganisms may even move down into the lungs, exchanging DNA so as to then emerge as Candida spp. a frequent asymptomatic colonizer of the upper respiratory tract, and especially emerging among hospitalized patients. Or, these same microbes may emerge as primary Candida pneumonia, “life-threatening in patients with cancer since it directly contributed to the death of 84% of the patients in the present series.” (Medicine (Baltimore), 1993 May;72(3):137-42; PMID: 8502166.) Or, in those individuals where an infected tooth or periodontal disease is present, the candida may move into the mouth and then, directly into the blood stream. Then the Candida readily attached to the plaque deposits in the arteries of the heart - where it often causes a fatal heart attack! As soon as it transforms and begins to put down its budding rhizoids, the finger-like roots break up plaque deposits that have been building up over the years and years. That causes a rapidly forming mass, and most persons are overwhelmed by the massive injury that results. I believe this rather clearly and simply explains why the incidence of heart disease is about twice as high in people with periodontal (gum) disease. Until this explanation came along, however, no plausible cause had ever been suggested. Other recent studies have indicated however, that the most common strain of bacteria in dental plaque also causes blood clots. So the all important thing is the rhizoid forming Candida that escape into the blood stream, and not the blood clots around a tooth.

This unseen relationship between increased heart attacks, candida and an infected tooth or periodontal diseases and other heart illnesses is also neatly explained in terms of the overt facts of history. In the not too distant past, the lessons of medical history show that while no one in previous generations had the ability to explain how great numbers of candida were what caused the toxins of “severe heartburn,” everyone was nevertheless were very much aware that “severe heart burn” frequently graduated to - “fatal heartburn!” Other researchers have also found that similar infections with Cytomegalovirus (CMV), (a usually dormant virus that can cause mononucleosis,) is associated with a higher risk of heart attacks or strokes. Other have found that infections with more than one of several common disease-causing germs can also increase the risk for heart attacks and strokes. They think people who had one or more of CMV, Chlamydia pneumonia (respiratory infections), Helicobacter pylori (ulcers), and hepatitis A (a liver disease,) were also at increased risk for heart attacks. Thus, people exposed to all four pathogens had a higher risk compared with those exposed to just one of the four, but I still contend that no one previously grasped the fact that heart attacks and strokes are mainly due to the sudden presence of drug resistant Candida with its ability to transform into a cell complete with rhizoids, after entering into the blood stream.

This leads us to another factor of these diseases, or what is known as “stress factors.” Every tissue has its own set of ecological values - which match those of the environment required by some type of bacteria. Furthermore, when the microbes involved are high-energy, drug resistant bacteria, in those cases where infection occurs, they grow in abundance and they commonly generate their own toxic byproducts - as does Candida and many other organisms. The presence of these microbes is what produces the so-called “stress factor.” It emerges, regardless of whatever tissue may become “infected” or otherwise involved.

Why doesn’t everyone know about all this? The answer is simply because no one has ever previously realized that cancers and the other “drug resistant diseases” can be viewed as a class, once one understands how they are caused by a specific class of very small bacteria which are able to transfer DNA into a living cell. Another real part of the overall problem has always been that these microorganisms are in fact, very difficult to see and seemingly impossible to isolate, because: a) the drug resistant strains are of an incredibly small size, and the transferable DNA’s that they exchange among themselves, or with other cells, are in fact, far, far smaller; b) they typically leave their ultra tiny, transferable DNA packets behind - which is readily proven by way of the fact that can only be seen by way of using an electron microscope accompanied by “metal staining techniques;” c) the presence of the unseen yet transferable DNA packets - identifiable only by using an electron microscope and metal staining techniques - actually explains why cancers and a large group of other drug resistant diseases reoccur so rapidly; d) the transferable packets of DNA modules also explains why this class of diseases (i.e., Cancers and The DNA Transfer Diseases,) are so difficult to medicate or otherwise eliminate; e) the drug-resistant bacteria that causes The DNA Transfer Diseases, typically grow very slowly under laboratory conditions, i.e., far to slowly to be readily recognized and/or discovered by using ordinary laboratory techniques; f) the phases of these organisms change when isolated in the laboratory, and most of these DNA transferring, drug resistant species will only grow at or near 37 degree C, (or only at average body temperature,) g) any cooler temperatures or normal atmospheric oxygen will readily prohibit the proper drug-resistant phase from being cultured and/or otherwise detected; h) they will grow in any normal laboratory where they are exposed to normal atmospheric oxygen, i) and they tend to only grow and reproduce (at normal body temperatures) and they are active only when oxygen levels are low. In addition to the evidences available showing the existence of the transferable packets of DNA by electronic microscope and metal staining techniques, all cancers and tumors can be neatly categorized and otherwise explained in terms their being either: 1) tumors, which usually grow very slowly and result from a DNA packet that will only replicate within a given, narrow range of activity - so as to produce only some very limited variation of the same cell, over and over again; or 2) neoplastic cancers, the result of a transferable DNA packet that has the DNA modules within it to readily change cell features, and they thereby replicate along a very broad range of activity; and 3) hormone sensitive cancers, which are driven by a transferable DNA packet that contains some form of normal hormonal features. Hence, the importance of the previous note that some bacteria in the clostridium family break down fats into the minor estrogens, otherwise known as “impeded estrogens.” In other words, when the otherwise foreign DNA from a drug-resistant clostridium gains entry into a breast cell for example, that cell becomes cancerous, it rapidly divides, and the every growing mass of cells will produce the same “impeded estrogen.”

Their growth conditions are so extreme that one such cancer causing, DNA transferring bacteria that has already been found, turned out to be so unusual that they in some ways approximate the growth conditions of Campylobacter. After more than a year of patient investigation, when researchers had attempted time and time again to isolate one such microorganism, often found to be closely associated with Candida, they only succeeded when cultures were inadvertently left in a sun warmed incubator, or apparently in a non-air conditioned laboratory, for a period of 6 days over the Easter holidays! This chance prolongation of temperatures and the incubation period (from the usual 2 days to 6 days!) resulted in the successful growth and isolation of a microorganism once described in 1925 in Nature as being “obtained almost constantly from cancers.” That same bacteria is now better known as Helicobacter pylori. In 1925, James Young wrote on it . . . “Of all the diseases of man cancer has probably the most unique features; it has little direct contact at any point with other morbid processes, and, for that reason, analogy is of small positive help in a search for the cause. The essential fact which marks it off, is that, beginning in the proliferation of a small group of cells, it ends, when running its ordinary course, as a myriad host of anarchic cells destroying by direct inroad the healthy surroundings and burrowing often into the springs of life itself. Any class of cells in the body may be seized with this tragic impetus, and, according to their site of origin, so will the cancer differ in structure and, it may be, in deadliness; but in all, no matter what the origin, the same unique features dominate the process, in all we have a plague of riotous, cells spreading as a blight without control or cessation.”

When tested for their cancer causing properties, the 20-year old archived blood samples of patients in a large sample who had eventually developed gastric malignancy were tested for antibodies to H. pylori, and compared to samples of an otherwise similar, but cancer free control group. Statistical analysis of the data indicated that those infected with H. pylori at the time the blood was collected were up to six times more likely to have subsequently developed a malignancy. At that point, the connection of H pylori to stomach cancer became so certain that the World Health Organization International Agency for Research in Cancer classified it as a class I carcinogen. (Lynch, Nancy A., “Helicobacter pylori and Ulcers: a Paradigm Revised, University of Iowa College of Medicine, Department of Pathology.) Another way to put it is, at one time, before the process of homogenization was invented, stomach cancers were the leading cause of death in America. It is fairly obvious at this point that the milk fats in some instances contained bacteria from the clostridium family – and the transferable DNA from these milk fats were what was causing all the stomach cancers, as well as directing the normal break down of milk fats into the minor estrogens.

All I am saying is that, just as stomach cancers were once caused by drug resistant Clostridium and mainly by Helicobacter pylori now, and H. pylori IS AN INCREDIBLY DIFFICULT BACTERIA TO ISOLATE AND DECTECT – YET IT HAS THE ABILITY TO TRANFER DNA - which is known to result in rapidly growing, drug-resistant, stomach malignancies, etc., then there are of course, other drug resistant bacteria WITH THE SAME ABILITY TO TRANSFER DNA,” which are of course responsible for the many other cancers and tumors. In brief, this means that every oncogene now listed by various scientists around the world, is actually composed of one of these little packets of transferable DNA’s, and that cell-directing material is simply being integrated into a given cell, once that particular cell divides. To be strictly scientific, this explains each oncogene whether it refers to a cancer causing oncogene or not. When the cell becomes cancerous the researchers of today are saying that the cell’s genes are “potentially oncogenes” and the term has thus shifted from oncogene to protooncogenes. It doesn’t matter what it’s called, simply put, the transfer of drug resistant DNA causes the cells genes to become cancerous – by causing any of a variety of changes, such as over-expression, under-expression, mutation, inactivation, differing substrates, different affinity for substrate, etc. and so this identifies were all the various oncogenes come from. In other words, every tissue has its’ own ecological values - which match those of the environment required by certain strains of the various types of DNA transferring bacteria, and this is the basic cause of cancers, tumors, and each of a long list of other, gene related diseases.

Note: Submitted on Nov 3, 2003, by Bruce D. McKay

This is all original research by Bruce D. McKay. It is the result of a thirty year epistemological study of oncology and the biological sciences. McKay who just turned 62 year old, has spent most of his adult life as a biological epistemologist. He can be reached for further comment, or other information etc., at: 11308 N. Hamner Ave. in Tampa, FL, 33612.
Mr. McKay can also be reached at: (813) 933-4905.

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