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Hello there,

Please dont give up because you are diagnosed. I think you doing the right thing looking for alternatives. Best way as far as I can see would be to try to keep viral load down as best as you can by various therapies: water, fruits and nuts, coconut oil, ACV like you are considering. Also though information on Miracle-Mineral-Supplement and HIV are differing, it wont hurt to give it a try.

If you should ever consider herbal anti-retrovirals, consider this supplier in India: hootone,org I know people back in Ghana where these products have helped a lot. Their products can bring viral load to undetectable and after prolonged use you could be cured completely. It could be expensive though depending on how you look at it. One month treatment is $100 plus shipping. Also if you have money to spend travel to Ghana and in 90 days you could be completely cured and everything. And trust me these two sources are genuine. Visit
http://www.mywebsiteafrica.com/alternativehivmed


I wish you the very best.

Otimus

Hi Sara,
I just tried to send you a personal message, and was unable to. Would you change your settings to allow personal messages? Thanks!

Sorry, but the Sutherlandia study showed no such thing. Here's my analysis on it (posted again):

I just read the study on which the OPC herbal cancer cure Sutherlandia success is based (available here at

http://www.tbyil.com/HIV_OPC_Trial.pdf

for anyone else who wants to take a look. I hope other people will look at the data and let me know what they think).
There are a few problems with seeing this study as proof that sutherlandia works.
The first problem is that the randomization went awry. The placebo and treatment groups started off with CD4+ T-cell counts way more different than could be expected by chance (average of 338 vs. 220.3). Using a 2-sample t-test, the probability of getting a difference this large by chance is well under 1%.
A more striking example of the weirdness of this study is that the placebo group experienced far more severe CD4 T-cell declines than one would expect if they were not taking any drugs at all. Different studies show slightly different estimates of the rate of CD4 decline in the absence of effective treatment, but with the exception of the more rapid CD4 decline in individuals who stop taking antiretroviral drugs, the decline is not nearly as dramatic as the people in this study taking the "placebo" experienced. All of the people in the placebo group who completed treatment had initial CD4 counts of between 200 and 400. Their CD4 counts decreased an average of 87 cells/ml in 2 months! A study of CD4 decline in South Africans showed average annual CD4 declines of only 30.6 cells/ml in those with starting CD4 counts of 351-500, and only 20.5 cells/ml in those with baseline counts of 201-350. If one is taking the data in the Sutherlandia study at face value, it is almost impossible to come to any other conclusion than that the "placebo" (an herb cocktail without Sutherlandia) harmed those in the placebo group (since they experienced a CD4 cell decline of about 40 times what would have been expected in a 2-month period had they received no treatment; we all already know that natural herb cocktails can be harmful as well as helpful). When the placebo is itself causing harm well beyond that which could be expected with no treatment, it really is not a placebo-controlled trial. The effects of the Sutherlandia are therefore unclear (just as it would be unclear if I claimed that a common poison antidote was beneficial for cancer patients, and tested it giving one group a "placebo" that was the poison, and the other group the poison plus the antidote. More people would die in the poison group, but that would not indicate that the antidote did anything for cancer!)
But still, the treatment group not only did better in terms of CD4 count than the placebo group did; they experienced INCREASES in CD4 counts. I would caution anyone from thinking that short-term CD4 increases are equivalent to true immunologic recovery, or that they necessarily lead to a good outcome for the patient. Many things have been shown to produce short-term CD4 increases, and yet are not generally considered helpful for HIV patients because they do not improve long-term outcomes. Examples would include AZT monotherapy, prednisone, and Interleukin-2 (IL-2; an interesting little cytokine - this is the synthetic version of the stuff your body makes naturally; it has its uses in some patients with an immunologic recovery discordant with their virologic suppression, but does not produce better outcomes in the general HIV population). Without adequate virologic suppression, producing more CD4 cells that are not HIV-specific may even "feed the fire" by giving the virus more cells that it can productively infect.
A 2-month period is far too short for this sort of study. AZT monotherapy worked wonderfully in the short term! This study did not measure viral load. For this reason, it is impossible to know whether the compound under study actually suppressed the virus, or whether it elevated CD4 counts by some other mechanism (which may not be helpful). Supposing that the Sutherlandia DID suppress the virus (which was not shown, even though it would have been very easy to test for viral load at the same time that CD4 counts were tested), long experience with this virus shows that it can mutate so as to escape any single inhibiting compound that you throw at it. 2 months is simply not long enough to see the long-term effects.

I think this is an interesting (albeit horribly performed) study. If someone wishes to do a decent study on this compound, here is what I would recommend:
- 4 arms to the study: one group receiving the Sutherlandia; one group receiving the "placebo" herbal treatment; one group receiving nothing; one group receiving antiretroviral therapy
- measure viral load as well as CD4 increases; if feasible, also test the CD4 cells in the groups reporting gains to see whether the new CD4 cells are HIV-specific
- follow the patients for at least 2 years
- larger numbers of patients would be preferable, but a small study is acceptable as a first step

If a well-performed, randomized placebo-controlled study showed that Sutherlandia produced long-term improvements without toxicity, I think everyone involved in this fight against HIV/AIDS would be very excited! Unfortunately, this study shows no such thing. I hope that someone will take this challenge on, and I also hope that it produces good results.

I would love to hear anybody else's views about this study.

for what it's worth, the study also makes many claims about the implications of its data that simply are not supportable. For instance, its author states that it disproved the null hypothesis (the null hypothesis states that the treatment had no effect). This is a statistical argument. As anyone who has taken even the most basic statistics course knows, you can never prove a null hypothesis incorrect, no matter how strong the evidence against it is. Statistical analysis can provide EXTREMELY strong evidence against the null hypothesis, but this can only make you confident to some degree less than 100% that the null hypothesis is incorrect. Perhaps the most ridiculous thing about this statement is that the author did no statistical analysis to ensure that the results were beyond that which would have been expected by chance (incidentally, they were - I ran a t-test, but this is just about meaningless since the randomization didn't work, and the placebo seems to have had effects on its own). To show that the author's statement of the null hypothesis is silly, consider if I did a trial where 56% of people on treatment improved, and 55% of people on placebo improved, without any statistical analysis, I could say "look! treatment is better than placebo! I disproved the null hypothesis that they would be the same!" This discounts the effect of chance variation - the possibility that I got people in the treatment group who would have gotten better no matter what. Statistical significance tests can make you "confident" that your results are not due to chance variation. The author performed none of these. I have no idea why the author is using the terminology of statistics without any of the actual statistics that could back him up. The use of the term "null hypothesis" makes no sense in this context.

Another misstatement: "Although the study was conducted over a relatively short period and with a small group of participants, the overwhelmingly positive results show that one should expect to get the same outcome over a longer period and with more participants." No! You cannot generalize the data beyond what it shows! In the context of HIV, one should absolutely not expect to get the same data in the long-term. Too much reearch has shown otherwise. That's why you actually have to do long-term research on clinical outcomes.

The author's conclusion is stated as "The following categorical statement can thus be made: An herbal mixture of Sutherlandi Frutescens and Oleander Nerium is
effective in increasing the CD4 counts of HIV-positive individuals with
initial CD4 counts of less than 400 in a meaningful way over a 60-day
period." Researchers should be very careful about making "categorical statements", especially based on a study as small and flawed as this one. Even if it were not preceded with this phrase, the "in a meaningful way" is yet to be determined. The author did not even do tests to show that the CD4 increase was statistically significant, much less that the statistically significant short-term increase is "meaningful".

The author appeared to come into this study with a bias against the belief that HIV alone can cause AIDS. He is allowed to believe whatever he wants; what he should not do is misinterpret the results to support this conclusion. He states that if Sutherlandia works, this supports the theory that there may be causes of AIDS other than HIV, because it shows that compounds other than antiretroviral drugs can work. This is unsupported by this paper: even if Sutherlandia showed real, long-lasting results (as yet unproved), this would not support the co-factor hypothesis because for all we know, Sutherlandia IS a compound with antiretroviral effects!

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